Claudia Ida Brodskyn
Português, Brasil
Graduada em Biomedicina pela Escola Paulista de Medicina (UNIFESP). Fez Mestrado e Doutorado em Imunologia pela USP e possui pós-doutorado realizado na CSU-Fort Coliins-Colorado-EUA. Atualmente, é Pesquisadora Titular do Centro de Pesquisa Gonçalo Moniz -FIOCRUZ-BA e Professora Adjunta da UFBA. Tem experiência na área de Imunologia, com ênfase em Imunoparasitologia, atuando principalmente nos seguintes temas: vacinas contra leishmaniose e imunorregulação na leishmnaniose humana e experimental. Pesquisadora 1B do CNPq.
Titulação:
Doutorado em Imunologia, USP
Vínculo:
Permanente
Email:
Telefone:
071.31762264
Currículo Lattes:
Orientando:
Produção Científica:
- Leukotriene B licenses inflammasome activation to enhance skin host defense.
- Unbalanced Production of LTB 4/PGE 2 Driven by Diabetes Increases Susceptibility to Cutaneous Leishmaniasis.
- Autophagic Induction Greatly Enhances Leishmania major Intracellular Survival Compared to Leishmania amazonensis in CBA/j-Infected Macrophages.
- Biomarkers in Zoonotic Visceral Leishmaniasis.
- Circulating Biomarkers of Immune Activation, Oxidative Stress and Inflammation Characterize Severe Canine Visceral Leishmaniasis - 2016
- Clinical and immunopathological findings during long term follow-up in Leishmania infantum experimentally infected dogs.
- Coinfection With Trypanosoma brucei Confers Protection Against Cutaneous Leishmaniasis.
- Degranulating Neutrophils Promote Leukotriene B4 Production by Infected Macrophages To Kill Leishmania amazonensis Parasites - 2016
- Dendritic Cells and Leishmania Infection: Adding Layers of Complexity to a Complex Disease - 2016
- Differential expression of the eicosanoid pathway in patients with localized or mucosal cutaneous leishmaniasis - 2015
- Granzyme B Produced by Natural Killer Cells Enhances Inflammatory Response and Contributes to the Immunopathology of Cutaneous Leishmaniasis.
- Granzyme B Produced by Natural Killer Cells Enhances Inflammatory Response and Contributes to the Immunopathology of Cutaneous Leishmaniasis.
- Heme Drives Oxidative Stress-Associated Cell Death in Human Neutrophils Infected with Leishmania infantum.
- Immunization of experimental dogs with salivary proteins from Lutzomyia longipalpis, using DNA and recombinant Canarypox virus induces immune responses consistent with protection against Leishmania infantum.
- Immunization with LJM11 salivary protein protects against infection with Leishmania braziliensis in the presence of Lutzomyia longipalpis saliva.
- Inflammasome activation by CD8+ T cells from Patients with Cutaneous Leishmaniasis caused by Leishmania braziliensis in the immunopathogenesis of the disease..
- Integrated analysis reveals that miR-193b, miR-671 and TREM-1 correlate with a good response to treatment of human Localized Cutaneous Leishmaniasis caused by Leishmania braziliensis.
- Lutzomyia longipalpis Saliva Drives Interleukin-17-Induced Neutrophil Recruitment Favoring Leishmania infantum Infection.
- Lutzomyia longipalpis Saliva Induces Heme Oxygenase-1 Expression at Bite Sites.
- Molecular Aspects of Dendritic Cell Activation in Leishmaniasis: An Immunobiological View.
- Natural infection by Leishmania infantum in the Lutzomyia longipalpis population of an endemic coastal area to visceral leishmaniasis in Brazil is not associated with bioclimatic factors.
- New Role of P. brasiliensis α-Glucan: Differentiation of Non-conventional Dendritic Cells.
- Plant-feeding phlebotomine sand flies, vectors of leishmaniasis, prefer Cannabis sativa.
- Poly(anhydride) nanoparticles containing cashew nut proteins can induce a strong Th1 and Treg immune response after oral administration.
- Vaccination with Leishmania infantum Acidic Ribosomal P0 but Not with Nucleosomal Histones Proteins Controls Leishmania infantum Infection in Hamsters - 2015
